Answer:
The Hay-flick Limit is a concept that helps to explain the mechanisms behind cellular aging. The concept states that a normal human cell can only replicate and divide forty to sixty times before it cannot divide anymore, and will break down by programmed cell death or apoptosis.
Answer:
Deep
Explanation:
Answer:A
Explanation: CAPPING
2. Can a bird have a house?
3. Can a bird have a truck?
Answer: The Leber's hereditary optic neuropathy (LHON) is caused by defective genes contained in mitochondrial DNA.
Explanation:
LHON is causes the vision loss. This occurs due to mutation in the mitochondrial DNA. The symptoms are not associated vision loss thus it is difficult to predict that which member of family was the carrier of the disease. Some people do not get completely blind. A young male is expected to develop a risk of 50% development of this disease.
The best way of solving this is to draw a Punnett square.
You know the F0 had one parent with singed bristles (s), and normal wings (L), and the other parent is normal bristles (S) with vestigial wings (l).
If you do the cross ssLL x SSll you'll find 100% of the offspring is F1: SsLl, this means, all of them show the dominant traits: normal wings and normal bristles.
If you cross two parents from F1 to have F2, you'll find:
SsLl x SsLl = SSLL + SslL + sSlL+ ssll = 25% SSLL,all dominant traits. 50% SsLl is a recessive trait carrier but shows dominant traits. 25% ssll this one has all recessive alleles, which means, it will show vestigial wings and signed bristles.
The answer is there is no specific time limit.
Residual condition refers to the condition, which remains after the end of the acute phase of an injury or illness. It can also be described as a later effect occurrence. The residual conditions may take place at any time after an illness or an acute injury.
The late effect or residual conditions may include burn, abscess, crushing injury, tuberculosis, poliomyelitis, and fracture.
Following are the steps of the signal transduction in order from the circulation in the blood to teh alteration of the cell's metabolism.
1. Insulin circulates in the blood.
2. Insulin combines extracellularly with the insulin receptor and stimulates receptor tyrosine kinase.
3. The receptor tyrosine kinase phosphorylates another tyrosine kinase.
4. Proteins, which comprise of a SH2 domain determine the stimulated tyrosine kinase.
5. SH2 comprising proteins are stimulated by the phosphorylated receptor tyrosine kinase.
6. Alteration of cell metabolism takes place.