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Explanation:
i didnt pay attention last year soooo sorry
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The length of the classroom is 10 meters and the width is 8 meters. The population size of the classroom is 40 students. What is the populiaotndensity of this classroom?
_____releases carbon dioxide as a waste product. *ChloroplastO CytoplasmRibosomesO Mitochondria
What would be the appropriate range for resistance and repetitions when one is designing a resistance training program targeted to develop strength? Muscular endurance? Muscular power? Hypertrophy?
Which list of words describes an action potential?all or none, neurotransmitters, membrane potentialmembrane potential, neurotransmitters, electrical signalselectrical signals, all or none, neurotransmittersall or none, membrane potential, electrical signals
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An ecosystem is a geographic area where plants, animals, and other organisms, as well as weather and landscape, work together to form a bubble of life
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The chance that the child produced would have nail-patella syndrome from this mating is 1/8 (12.5%) and the chance that the child has alkaptonuria is zero (0%). It is a case of recessive phenotypes.
Pedigree and genetic inheritance
The answer is incomplete, but I have now included the Figure to answer it. The vertical lines indicate nail-patella syndrome, while horizontal lines indicate alkaptonuria.
Pedigree is a diagram showing the genealogical relationships in different individuals, which is usually used to represent the inheritance pattern of a given phenotype.
In this case, the nail-patella syndrome and alkaptonuria are recessive phenotypes, which means that individuals may be heterozygous carriers but they need to have both mutated alleles to express each condition.
In the pedigree, it is possible to observe that the individual IV5 expresses both syndromes (nail-patella syndrome and alkaptonuria), whereas there is a 1/8 chance that the individual IV2 is a carrier of the nail-patella syndrome.
Learn more about pedigree here:
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The mass extinction that led to the loss of 96% of marine life and 70% of terrestrial life was most likely due to a supernova, an asteroid impact or Volcanic Eruption.
What is Mass extinction?
An extinction event is defined as a widespread and rapid decrease in biodiversity on Earth. Those events are characterized by sharp changes in the diversity and abundance of multicellular organisms that occur when extinction rates increase relative to background extinction rates and species evolution rates.
This mass extinction which happened during the end of the Permian era. The extinction of 96 percent of all marine species and 70 percent of all terrestrial species resulted in catastrophic changes on Earth, which became almost devoid of all species.
The cause of this mass extinction is related to volcanic activity, supernova or asteroid impact which could have increased the average temperature of the Earth's surface.
Thus, the mass extinction that led to the loss of 96% of marine life and 70% of terrestrial life was most likely due to a supernova, an asteroid impact or Volcanic Eruption.
Learn more about Mass Extinction, here:
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Answer:
due to volcanic eruption
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Answer:
Mixed inhibition refers to the combination of two reversible types of enzyme inhibition, competitive inhibition and non-competitive inhibition. The term mixed is used when the inhibitor can bind both the free enzyme and the enzyme-substrate complex. In mixed inhibition the inhibitor is in a different place from the active site where the substrate is found.
Mathematically, mixed inhibition occurs when both alpha and alpha-prime factors (introduced in the Michaelis-Menten equation representing competitive and non-competitive inhibition respectively) are present (they are larger than unity).
In a special case of mixed inhibition, the alpha and alpha-prime factors are the same, then non-competitive inhibition occurs.
With this type of inhibition Km depends on the affinity of the inhibitor to join E or ES and Vmax decreases.
Enzymatic inhibitors are molecules that bind enzymes and decrease their activity. Since blocking an enzyme can kill a pathogen or correct a metabolic imbalance, many medications act as enzyme inhibitors. They are also used as herbicides and pesticides. However, not all molecules that bind to enzymes are inhibitors; Enzymatic activators bind to enzymes and increase their activity.
The binding of an inhibitor can prevent the substrate from entering the active site of the enzyme and / or hinder the enzyme from catalyzing its corresponding reaction. The inhibitor binding may be reversible or irreversible. Normally, irreversible inhibitors react with the enzyme covalently and modify their chemical structure to the level of essential residues necessary for enzymatic activity. In contrast, reversible inhibitors bind to the enzyme in a non-covalent manner, resulting in different types of inhibitions, determined whether the inhibitor binds to the enzyme, the enzyme-substrate complex or both.
Many medications are enzymatic inhibitors, so their discovery and improvement is an active field of research in biochemistry and pharmacology. The validity of a medicinal enzyme inhibitor is usually determined by its specificity (its inability to bind to other proteins) and its potency (its dissociation constant, which indicates the concentration necessary to inhibit an enzyme). A high specificity and potency ensures that the medication will have few side effects and therefore a low toxicity.
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The function of the Calvin cycle is to take CO2 from the air and bond it together to form the molecules that will make the sugar glucose. This process is called carbon fixation . The actual result of the Calvin cycle is not glucose, a 6-carbon sugar, but another molecule called G3P. G3P is an abbreviation for glyceraldehyde-3-phosphate. This molecule is later converted by other chemical reactions into glucose.
It was in my lesson.