The right answer is 5.
To answer this question, we must think in the same way as the demonstration showing that codons of 3 are necessary to encode an amino acid (in terrestrial species).
To calculate the number of nucleotides in a codon in these new species: we should use the law of:
nucleotides existing ^ (number of nucleotides in a codon) = number of codons. (in order to have the number of possible codon combinations)
The number of codons must be greater than or equal to the number of amino acids (17).
2 ^ 1 = 2 (<17)
2 ^ 2 = 4 (<17)
2 ^ 3 = 8 (<17)
2 ^ 4 = 16 (<17)
2 ^ 5 = 32 (> 17)
32> 17 so the number of nulceotides in a codon must be 5.
The minimum size of a codon for the hypothetical Martian life-forms, which use only two bases (A and T) and have only 17 amino acids in their proteins, should be five bases. This size is needed to generate a minimum of 17 different codons.
The question is asking for the minimum size of a codon in these hypothetical Martian life-forms that use only two bases (A and T) in their DNA and have only 17 amino acids in their proteins. To analyze this, we must comprehend how codons function. A codon is a sequence of nucleotides in RNA that codes for a specific amino acid in a protein. In life on Earth, each codon is made up of three bases (each base can be A, T, C, or G), which makes for a total of 4^3 = 64 possible codons coding for 20 different amino acids.
However, in the Martian organisms, there are only two available bases (A and T) for constructing codons. In order to generate at least 17 different codons to code for the 17 different Martian amino acids, each Martian codon would have to be made up of at least 5 bases (since 2^4 = 16 is not enough, and 2^5 = 32 would be sufficient).
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tRNA
tRNA synthetase
promoter
allele
nucleic acid
Answer:
I think its tRNA
Explanation:
Correct answer: A). Club
Club fungi are heterotrophic and it can be saprophytes. The enzyme present in the club fungi decompose the leaves, wood and other organic materials. It can be eaten by other fungi and bacteria.
It is distinguished from the other fungi by the presence of a club-shaped organ called as a basidium. Puffballs, mushroom and shelf fungi are the members of this group.
Hence, the correct answer would be option A.
Most jellyfishes use a form of jet propulsion to move through seawater. ... This is what most jellyfish do. They squeeze their bodies in order to push jets of water from the bottom of their bodies to propel the jellyfish forward. Now check out another method of jellyfish propulsion, used by the comb jellyfish.
Hope this helps :D
The development of different species are similar.
The DNA of different species are similar.
Experiments can form the molecules of life out of chemicals.
Fossils show a history of life that gets more complex.
Fossils show different stages of evolution in a species.
The fossil record is backed up by the geological record.
Answer:
I think that the answer is 3567
The equivalent energy of an object given its mass , through the equation,
E = mc²
where c is the speed of light (3 x 10^8 m/s)
Substituting the known values,
E = (1.05 g/ 1000) (3 x 10^8 m/s)²
E = 9.45x10^13 J
Thus, 9.45x10^13 J is answer.
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The glomerular lesion that is characterized by thickening of the glomerular capillary wall with immune deposition is called membranous glomerulonephritis.
In this condition, there is a buildup of immune complexes, which are composed of antibodies and antigens, in the glomerular capillary walls. This immune deposition leads to inflammation and thickening of the walls, impairing the filtration function of the kidneys.
The thickening of the glomerular capillary wall in membranous glomerulonephritis is caused by the accumulation of immune complexes in the subepithelial space. These immune complexes can be formed due to various underlying causes, such as autoimmune diseases, infections, medications, or certain cancers. The immune complexes trigger an inflammatory response, attracting immune cells to the glomerular capillary walls and causing damage.
As a result of the thickening of the glomerular capillary wall, the normal filtration process in the kidneys is disrupted. This can lead to proteinuria (presence of excess protein in the urine), hypoalbuminemia (low levels of albumin in the blood), and edema (swelling) in the affected individual. Over time, the chronic inflammation and scarring can further impair kidney function and may progress to chronic kidney disease.
To diagnose membranous glomerulonephritis, a kidney biopsy is typically performed to examine the glomeruli under a microscope. The presence of thickened glomerular capillary walls with immune deposits confirms the diagnosis.
Treatment for membranous glomerulonephritis aims to reduce inflammation, control symptoms, and preserve kidney function. Depending on the underlying cause and severity of the condition, treatment options may include medications such as immunosuppressants, angiotensin-converting enzyme inhibitors (ACE inhibitors), or angiotensin receptor blockers (ARBs). In some cases, individuals may require supportive measures such as dietary modifications and fluid restriction.
In summary, membranous glomerulonephritis is a glomerular lesion characterized by thickening of the glomerular capillary wall due to immune deposition. It can result in kidney dysfunction and is typically diagnosed through a kidney biopsy. Treatment options focus on reducing inflammation and preserving kidney function.
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